Role of the Jewish physician in covering up TB Infections among Russian Jewry

[CrackSmokeRepublican]

I am of the firm belief that Jews carried strains of mycobacteria with them over hundreds of years, the type that leads to the infection of TB/Diabetes. They brought this out of their ghettos and perpetuated it in the 1800s from their typical trade in reclaimed (tubercular) animal skins. While Jews have carried a genetic resistance to TB from the ghettos of Europe, this did not stop them from carrying TB mycobacteria outside of their Ghettos and Shetls and into "Goyim" cities.  It is interesting that from the time of Jewish liberation by Napoleon in the early 1800s until the 1920s, a tremendous rise in TB occurred in both Europe and America -- a period that coincidentally mirrored Eastern European Jews leaving the Ghettos of the Pale of Settlement.  

The documented "Resistance" of Jews to TB is due to their age old and well documented unhealthy behaviors in Europe such as not washing or removing lice infections.  These immigrants were used as a weapon to infect TB (Diabetes?) upon Western Goyim nations when they arrived upon foreign shores, IMHO.  Schizophrenia is also a well known Jewish disease often carried by Jews which has been transferred to Goyim people in modern times.

Historically, Ancient Egyptians (with mostly Western DNA) removed the Jews from Egypt due to "Diseases" -- perhaps TB, Schizophrenia, Diabetes, Lice were among those diseases?  

For example, here is a typical Tubercular Jew serving in the Czar's Army that came to America:
http://www.aiisf.org/index.php/immigran ... and-miriam
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This professor writing at the turn of the century named Spivak tried to portray Jews as somewhat "healthy" compared with other immigrants... perhaps it is just the old "Kol-Nidre" at work? Jews made huge mint off of treating TB as they infected others with it... --CSR



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Synopsis

Part biography, part medical history, and part study of Jewish life in turn-of-the-century America, "Miracle Man" tells the story of Dr Charles David Spivak - the Jewish immigrant from Russia who became one of the leaders of the American tuberculosis movement. Born in Russia in 1861, Spivak immigrated to the United States in 1882 and received his medical degree from Philadelphia's Jefferson Medical College by 1890. In 1896, his wife's poor health brought them to Colorado. Determined to find a cure, Spivak became one of the most charismatic and well-known leaders in the American tuberculosis movement. His role as director of Denver's Jewish Consumptives' Relief Society sanatorium allowed his personal philosophies to strongly influence policies. His unique blend of Yiddiskeit, socialism, and secularism - along with his belief in treating the 'whole' patient - became a model for integrating medical, social, and rehabilitation services that was copied across the country. Not only a national leader in the crusade against tuberculosis but also a luminary in the American Jewish community, Dr Charles Spivak was a physician, humanitarian, writer, linguist, journalist, administrator, social worker, ethnic broker, and medical, public health, and social crusader. Abrams' biography will be a welcome addition to anyone interested in the history of medicine, Jewish life in America, or Colorado history.

http://www.waterstones.com/waterstonesw ... k/7377975/

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Professor's book examines role of Jewish physician in early treatment of TB

While it's common knowledge that "gold-fever" attracted settlers to Colorado in the 1800s, few people realize that an entirely different [...]

By: Staff

May 4, 2009

Patients at the Jewish Consumptive Relief Society (JCRS) received heliotheraphy, or maximum exposure to the sun, believed to be an effective treatment for tuberculosis.

While it's common knowledge that "gold-fever" attracted settlers to Colorado in the 1800s, few people realize that an entirely different sort of affliction contributed to the state's early growth, according to DU's Jeanne Abrams, a professor of Judaic studies.

Colorado's fresh, dry air, frequent sunshine and high altitude made it a natural haven for tuberculosis sufferers in the late 19th century. Although Robert Koch identified the bacteria responsible for tuberculosis — also known as consumption or the white plague — in 1882, many physicians still considered the 'proper environment' to be an effective weapon against the disease.

In her new book, Dr. Charles David Spivak: A Jewish Immigrant and the American Tuberculosis Movement (University Press of Colorado, 2009), Abrams recounts the story of Spivak, a philanthropist who took the socialist ideals that contributed to his exile from Russia and applied them to his work as a physician treating tuberculosis.

In the late 1800s, receiving treatment for tuberculosis was nearly impossible for members of the lower classes due to the high-cost of private sanatoriums and the absence of public hospitals. "The Jewish community was the first to step forward and fill this void," Abrams says, noting that a group of wealthy Reform Jews of German descent started the National Jewish Hospital for Consumptives in Denver in 1899.

Operating under the motto "None may enter who can pay — None can pay who enter," National Jewish offered treatment to anyone in the early stage of the disease free of charge.

As a socialist and traditional Jew, however, Spivak took issue with some of the hospital's policies, including their refusal to treat individuals with advanced tuberculosis and the absence of a kosher kitchen.

In 1904, Spivak, along with members of Denver's Jewish working class, created the Jewish Consumptives Relief Society (JCRS).

The JCRS, although formally nonsectarian, offered a more traditional environment for Orthodox patients and treated individuals in all stages of the disease, adhering to the motto: "He who saves one life saves the world."

Abrams has dedicated much of her career to providing a broader perspective of the American Jewish experience by researching Jewish history in the West.

"Many people think Jewish life only flourished on the East Coast," Abrams says. "It's been my mission to demonstrate that there was a vibrant Jewish community in the West."

Abrams' fascination with Spivak stems from research she conducted in the early 1980s on the JCRS, the subject of her doctoral dissertation, and from her previous books Blazing the Tuberculosis Trail (Colorado Historical Society, 1991) and Jewish Women Pioneering the Frontier Trail (New York University Press, 2006).

Like Abrams' past research, Dr. Charles David Spivak illuminates a neglected facet of history, American University history Professor Alan Kraut says.

"The history of medicine is often overlooked, and tuberculosis was a fierce and killer disease," Kraut says. "Spivak's story is in part an immigrant success story about a man with great social commitment who did a lot for the people he came in contact with. It's a very important contribution to social history and the history of social medicine."

http://blogs.du.edu/today/news/professo ... ment-of-tb

[CrackSmokeRepublican]

Here it is admitted Russian Jews carried TB to America while "hero" Jew Physician Spivak basically lied about it to Goyim America:


The classic case of heterozygote advantage in humans is sickle cell anemia, a disease for which carriers of several common mutations have greater resistance to malaria, an advantage in malarial environments. In the 1960s and 1970s, some researchers argued that there must be some evolutionary benefit to being a heterozygote for Tay-Sachs as well.[54][55]

In the 1970s and 80s, several researchers investigated whether being a Tay-Sachs carrier might have served as a form of protection against tuberculosis in medieval Europe. Tuberculosis was prevalent in the European Jewish populations, in part because Jews were forced to live in crowded ghettos. One statistical study has demonstrated that grandparents of Tay-Sachs carriers (who are more likely to have been carriers themselves) died proportionally from the same causes as non-carriers.[56]

A more recent theory of heterozygote advantage (attributed to Gregory Cochran) proposes that Tay-Sachs, and the other lipid storage diseases that are prevalent in Ashkenazi Jews, reflect genes that enhance dendrite growth and promote higher intelligence when present in carrier form. In this way, Cochran proposes that being a heterozygote provided a selective advantage at a time when Ashkenazi Jews were restricted to intellectual occupations.[57][58] (See Ashkenazi intelligence theory.)

Researchers of the 1960s and 1970s often favored theories of overdominance as an explanation of heterozygote advantage, but failed to find much evidence for them in human populations. They were also unaware of the diversity of the Tay-Sachs mutation base. In the 1970s, complete genomes had not yet been sequenced, and researchers were unaware of the extent of polymorphism. The contribution to evolution of genetic drift (as opposed to natural selection) was not fully appreciated.


Since the 1970s, DNA sequencing techniques using PCR have been applied to many genetic disorders, and in other human populations. Several broad genetic studies of the Ashkenazi population (not related to genetic disease) have demonstrated that the Ashkenazi Jews are the descendants of a small founder population, which may have gone through additional population bottlenecks. These studies also correlate well with historical information about Ashkenazi Jews. Thus, a preponderance of the recent studies have supported the founder effects theory.[37][38][39]


http://en.wikipedia.org/wiki/Tay%E2%80%93Sachs_disease

[CrackSmokeRepublican]

Researchers Say Intelligence and Diseases May Be Linked in Ashkenazic Genes -- Non Geneticists made this claim like "Collapse" author Jew Jared Diamond

By NICHOLAS WADE
Published: June 3, 2005

A team of scientists at the University of Utah has proposed that the unusual pattern of genetic diseases seen among Jews of central or northern European origin, or Ashkenazim, is the result of natural selection for enhanced intellectual ability.


Intelligence and Genetics

The selective force was the restriction of Ashkenazim in medieval Europe to occupations that required more than usual mental agility, the researchers say in a paper that has been accepted by the Journal of Biosocial Science, published by Cambridge University Press in England.

The hypothesis advanced by the Utah researchers has drawn a mixed reaction among scientists, some of whom dismissed it as extremely implausible, while others said they had made an interesting case, although one liable to raise many hackles.

"It would be hard to overstate how politically incorrect this paper is," said Steven Pinker, a cognitive scientist at Harvard, noting that it argues for an inherited difference in intelligence between groups. Still, he said, "it's certainly a thorough and well-argued paper, not one that can easily be dismissed outright."

"Absolutely anything in human biology that is interesting is going to be controversial," said one of the report's authors, Dr. Henry Harpending, an anthropologist and a member of the National Academy of Sciences.

He and two colleagues at the University of Utah, Gregory Cochran and Jason Hardy, see the pattern of genetic disease among the Ashkenazi Jewish population as reminiscent of blood disorders like sickle cell anemia that occur in populations exposed to malaria, a disease that is only 5,000 years old.

In both cases, the Utah researchers argue, evolution has had to counter a sudden threat by favoring any mutation that protected against it, whatever the side effects. Ashkenazic diseases like Tay-Sachs, they say, are a side effect of genes that promote intelligence.

The explanation that the Ashkenazic disease genes must have some hidden value has long been accepted by other researchers, but no one could find a convincing infectious disease or other threat to which the Ashkenazic genetic ailments might confer protection.

A second suggestion, wrote Dr. Jared Diamond of the University of California, Los Angeles, in a 1994 article, "is selection in Jews for the intelligence putatively required to survive recurrent persecution, and also to make a living by commerce, because Jews were barred from the agricultural jobs available to the non-Jewish population."

The Utah researchers have built on this idea, arguing that for some 900 years Jews in Europe were restricted to managerial occupations, which were intellectually demanding, that those who were more successful also left more offspring, and that there was time in this period for the intelligence of the Ashkenazi population as a whole to become appreciably enhanced.

But the Utah researchers' analysis comes at a time when some geneticists have suggested natural selection is not the reason for the Ashkenazic diseases after all. Two years ago, Dr. Neil Risch, a geneticist now at the University of California, San Francisco, proposed a different genetic mechanism known as a founder effect, which occurs when a population is reduced for a time.

He found that all the Ashkenazic diseases had similar properties, including having arisen within the last 1,100 years. Therefore they had all arisen through the same cause, he argued, which must be founder effects, because it was unlikely that all could be due to natural selection. Last year, Dr. Montgomery Slatkin of the University of California, Berkeley, came to much the same conclusion for different reasons.

The Utah team agrees with Dr. Risch that the diseases all arose in historical times from the same cause but say natural selection is more likely because none of the non-disease Ashkenazic genes they tested showed any sign of a founder effect. They say the clustering of four of the diseases in the same biochemical pathway could only have arisen under the influence of natural selection, and calculate that the odds of a founder effect producing such a cluster are vanishingly low.

The four diseases, all of which are caused by mutations that affect the cell's management of chemicals known as sphingolipids, are Tay-Sachs, Niemann-Pick, Gaucher, and mucolipidosis type IV. A second cluster of diseases affects repair of DNA.

Turning to the possibility that some infection was the cause of the selective effect, the Utah researchers noted that Ashkenazim and Europeans lived together in the same cities and were exposed to the same microbes. If disease were the agent of selection, the Utah team argues, the European population would have developed a similar genetic response.

Ashkenazi Jews occupied a different social niche from their European hosts, and that is where any selective effect must have operated, the Utah researchers say. From A.D. 800, when the Ashkenazi presence in Europe is first recorded, to about 1700, Ashkenazi Jews held a restricted range of occupations, which required considerable intellectual acumen. In France, most were moneylenders by A.D. 1100. Expelled from France in 1394, and from parts of Germany in the 15th century, they moved eastward and were employed by Polish rulers first as moneylenders and then as agents who paid a large tax to a noble and then tried to collect the amount, at a profit, from the peasantry. After 1700, the occupational restrictions on Jews were eased.

As to how the disease mutations might affect intelligence, the Utah researchers cite evidence that the sphingolipid disorders promote the growth and interconnection of brain cells. Mutations in the DNA repair genes, involved in second cluster of Ashkenazic diseases, may also unleash growth of neurons.

In describing what they see as the result of the Ashkenazic mutations, the researchers cite the fact that Ashkenazi Jews make up 3 percent of the American population but won 27 percent of its Nobel prizes, and account for more than half of world chess champions. They say that the reason for this unusual record may be that differences in Ashkenazic and northern European I.Q. are not large at the average, where most people fall, but become more noticeable at the extremes; for people with an I.Q. over 140, the proportion is 4 per 1,000 among northern Europeans but 23 per 1,000 with Ashkenazim.

The Utah researchers describe their proposal as a hypothesis. Unlike many speculations, it makes a testable prediction: that people who carry one of the sphingolipid or other Ashkenazic disease mutations should do better than average on I.Q. tests.

The researchers have identified two reasonably well accepted issues, the puzzling pattern of diseases inherited by the Ashkenazi population and the population's general intellectual achievement. But in trying to draw a link between them they have crossed some fiercely disputed academic territories, including whether I.Q. scores are a true measure of intelligence and the extent to which intelligence can be inherited.

The authors "make pretty much all of the classic mistakes in interpreting heritability," said Dr. Andrew Clark, a population geneticist at Cornell University, and the argument that the sphingolipid gene variants are associated with intelligence, he said, is "far-fetched."

In addition, the genetic issue of natural selection versus founder effects is far from settled. Dr. Risch, whose research supports founder effects, said he was not persuaded by the Utah team's arguments. Dr. David Goldstein, a geneticist at Duke University who was not connected with either Dr. Risch's or the Utah study, was more open on the issue, saying Dr. Risch had made "quite a strong case" that founder effects could be the cause, but had not ruled out the possibility of selection.

Dr. Slatkin, though favoring a founder effect over all, said he agreed with the Utah team that this would not account for the cluster of sphingolipid diseases.

As for the Utah researchers' interpretation of Jewish medieval history, Paul Rose, professor of Jewish studies at Pennsylvania State University, said, "I think that some of their conclusions may be right though they still need a lot of work to be persuasive to historians and others."

Dr. Gregory Cochran, the first author on the Utah team's paper and a physicist who took up biology, said he became interested in the subject upon learning that patients with a particular Ashkenazic disease known as torsion dystonia were told by their physicians that "the positive thing is that this makes you smart."

"When you're in a hurry and have strong selection, you have a lot of genes with bad side effects," he said. The Ashkenazi Jewish population seemed to fit this pattern, he said, since they married only inside the community, making selection possible, and they had an urgent need for greater intelligence. Evolution had therefore selected every possible mutation that worked in this direction, despite their harmful side effects when inherited from both parents. "In a sense, I consider this a very boring paper since it raises no new principles of genetics," Dr. Cochran said.

[CrackSmokeRepublican]

I'm of the opinion that Vivien Leigh's mental illness, promiscuity, and TB infection is related to her possibly having to sleep on a "casting couch" with one of the Selznick brothers in the late 1930s. Her life-long infection with Tuberculosis is likely related to her close relations with the Ashkenazi Jew Directors of Hollywood. I figure she went several years undiagnosed before the mid-1940s.  

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David O. Selznick





Early years

Selznick was born to a Jewish family in Pittsburgh, Pennsylvania, the son of silent movie distributor Lewis J. Selznick and Florence A. (Sachs) Selznick. Selznick added the "O" to his name later on a whim.[1]

He studied at Columbia University and worked as an apprentice for his father until the elder's bankruptcy in 1923. In 1926, Selznick moved to Hollywood, and with the help of his father's connections, got a job as an assistant story editor at Metro-Goldwyn-Mayer. He left MGM for Paramount Pictures in 1928, where he worked until 1931, when he joined RKO as Head of Production. His years at RKO were fruitful, and he worked on many films, including A Bill of Divorcement (1932), What Price Hollywood? (1932), Rockabye (1932), Our Betters (1933), and King Kong (1933). While at RKO, he also gave George Cukor his directing break. In 1933 he returned to MGM to establish a second prestige production unit, parallel to that of Irving Thalberg, who was in poor health. His unit's output included Dinner at Eight (1933), David Copperfield (1935), Anna Karenina (1935) and A Tale of Two Cities (1935).

Selznick International Pictures

Despite his successes at MGM, Paramount Pictures, and RKO Pictures, Selznick longed to be an independent producer with his own studio. In 1935 he realized that goal by forming Selznick International Pictures and distributing his films through United Artists. His successes continued with classics such as The Garden of Allah (1936), The Prisoner of Zenda (1937), A Star Is Born (1937), Nothing Sacred (1937), The Adventures of Tom Sawyer (1938), The Young in Heart (1938), Made for Each Other (1939), Intermezzo (1939) and Gone with the Wind (1939), which remains one of the all-time highest grossing films (adjusted for inflation). It also won seven additional Oscars and two special awards. Selznick also won the Irving G. Thalberg Memorial Award that same year.

In 1940, he produced his second Best Picture Oscar winner in a row, Rebecca, the first Hollywood production for British director Alfred Hitchcock. Selznick had brought Hitchcock over from England, launching the director's American career. Rebecca was Hitchcock's only film to win Best Picture.

Later productions

After Rebecca, Selznick closed Selznick International Pictures and took some time off. His business activities included the loan of his contracted artists to other studios, including Alfred Hitchcock, Ingrid Bergman, Vivien Leigh and Joan Fontaine. He also developed film projects and sold the packages to other producers. Among the movies that he developed but then sold were almost all of Hitchcock's films through to 1947, except for two that he released through Selznick International Pictures or Selznick Releasing Organization, Spellbound and The Paradine Case. In 1944 he returned to producing pictures with the huge success Since You Went Away, which he wrote. He followed that with Spellbound (1945), as well as Portrait of Jennie (1948), a vehicle for Jennifer Jones. In 1949, he co-produced the Carol Reed picture The Third Man with Alexander Korda.

Gone with the Wind overshadowed the rest of Selznick's career. The closest he came to matching it was with Duel in the Sun (1946) featuring future wife Jennifer Jones in the role of the primary character Pearl. With a huge budget, the film is known for causing moral upheaval because of the then risqué script written by Selznick. And though it was a troublesome shoot with a number of directors, the film would turn out to be a major success. The film was the second highest grossing film of 1947 and turned out to be the first movie that Martin Scorsese would see, inspiring the director's career.

"I stopped making films in 1948 because I was tired", Selznick later wrote. "I had been producing, at the time, for twenty years . . . . Additionally it was crystal clear that the motion-picture business was in for a terrible beating from television and other new forms of entertainment, and I thought it a good time to take stock and to study objectively the obviously changing public tastes . . . . Certainly I had no intention of staying away from production for nine years."[2] Selznick spent most of the 1950s nurturing the career of his second wife, Jennifer Jones. His last film, the big budget production A Farewell to Arms (1957) starring Jones and Rock Hudson, was ill received. But in 1954, he ventured into television, producing a two hour extravaganza called Light's Diamond Jubilee, which, in true Selznick fashion, made TV history by being telecast simultaneously on all four TV networks: CBS, NBC, ABC, and DuMont.
[edit] Personal life
Jennifer Jones and Selznick in Los Angeles, 1957.

Selznick married Irene Gladys Mayer, daughter of MGM mogul Louis B. Mayer, in 1930. They separated in 1945 and divorced in 1948.[3] They had two sons, Daniel Selznick and Jeffrey Selznick. He became interested in actress Jennifer Jones, who was then married to actor Robert Walker. They married in 1949. They had one daughter, Mary Jennifer Selznick, who committed suicide in 1976.

Selznick died in 1965 following several heart attacks, and was interred in the Forest Lawn Memorial Park Cemetery in Glendale, California. Until his death in 1944, Myron Selznick, Selznick's brother, was one of the most powerful agents in Hollywood.

 Legacy

Selznick had a keen instinct for new talent and brought Fred Astaire, Katharine Hepburn, Ingrid Bergman, Vivien Leigh, Louis Jourdan, and Alfred Hitchcock into the American film industry.

One example of his tendency to meddle is revealed in the book Memo From David O. Selznick, concerning the 1940 Hitchcock film Rebecca. Reading the screenplay submitted for his approval, Selznick was shocked to discover that director Alfred Hitchcock had allowed Daphne du Maurier's original novel to be changed so that it was virtually unrecognizable, even to the point of introducing unnecessarily comic scenes not in the book. The furious Selznick wrote Hitchcock a blistering memo, and forced Hitchcock to remain faithful to the novel. However, Hitchcock and the other screenplay writers rewrote the script in a way so Selznick would struggle when it was time to edit the film. Alfred Hitchcock's later film Spellbound was edited on Selznick's insistence, and the director grew resentful of his nature. Selznick also battled with Carol Reed during the production of The Third Man and re-edited the film for its American release. Perhaps the best known example of his interference was during the production of Powell and Pressburger's Gone to Earth starring his wife Jennifer Jones. After production, Selznick disliked the film and removed almost an entire third of it for its American release, under the title The Wild Heart with new material directed by Rouben Mamoulian. Selznick lost a court case with Powell & Pressburger to control all versions of the film but he retained control of the American release so he proceeded to cut and change various sections back in Hollywood.

Selznick may not have invented the "casting couch" but he tried to seduce every starlet that attracted him. He also used amphetamines constantly and smoked five packs of cigarettes a day. His incessant meddling in films which drove up their cost enormously may have been the result of the former habit (David Thomson Showman: The Life of David O. Selznick).

For his contribution to the motion picture industry, David O. Selznick has a star on the Hollywood Walk of Fame at 7000 Hollywood Blvd., in front of the historic Hollywood Roosevelt hotel.

Film library

After Selznick's death, his estate sold the rights to a majority of his post-1935 films to ABC (now part of The Walt Disney Company), although MGM bought in 1944 the rights to Gone with the Wind and, at some point, the 1937 version of The Prisoner of Zenda for its 1952 remake (all today part of the Turner Entertainment library owned by Time Warner), and 20th Century Fox still holds rights to the remake of A Farewell to Arms.

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Vivien Leigh

Lauded for her beauty, Leigh felt that it sometimes prevented her from being taken seriously as an actress. However, ill health proved to be her greatest obstacle. For much of her adult life Leigh had what is now known as bipolar disorder.[2] She earned a reputation for being difficult to work with, and her career suffered periods of inactivity. She also suffered recurrent bouts of chronic tuberculosis, first diagnosed in the mid-1940s. Leigh and Olivier divorced in 1960, and she worked sporadically in film and theatre until her death from tuberculosis in 1967.


Hollywood was in the midst of a widely publicised search to find an actress to portray Scarlett O'Hara in David O. Selznick's production of Gone with the Wind (1939). Leigh's American theatrical agent was the London representative of the Myron Selznick Agency (Myron was David's brother). In February 1938, Leigh asked that she be allowed to play Scarlett O'Hara. Selznick, who watched her performance that month in Fire Over England and A Yank at Oxford, thought her to be excellent but in no way a possible Scarlett, as she was "too British". Leigh travelled to Los Angeles to be with Olivier and to try to convince Selznick that she was Scarlett. When Myron Selznick, who also represented Olivier, met Leigh, he felt that she possessed the qualities his brother was searching for. Myron Selznick took Leigh and Olivier to the set where the burning of the Atlanta Depot scene was being filmed and introduced Leigh, telling his brother, "Hey, genius. Meet your Scarlett O'Hara." The following day, Leigh read a scene for Selznick, who organised a screen test and wrote to his wife, "She's the Scarlett dark horse and looks damn good. Not for anyone's ear but your own: it's narrowed down to Paulette Goddard, Jean Arthur, Joan Bennett, and Vivien Leigh". The director, George Cukor, concurred and praised Leigh's "incredible wildness"; she secured her role as Scarlett soon after.[19]

Filming proved difficult for Leigh. Cukor was dismissed and replaced by Victor Fleming, with whom Leigh frequently quarrelled. She and Olivia de Havilland secretly met with Cukor at night and on weekends for his advice about how they should play their parts. She befriended Clark Gable, his wife Carole Lombard, and Olivia de Havilland; but she clashed with Leslie Howard, with whom she was required to play several emotional scenes. Leigh was sometimes required to work seven days a week, often late into the night, which added to her distress; and she missed Olivier, who was working in New York. She said to Laurence Olivier on a long-distance call, " Puss, my puss, how I hate film acting! Hate, hate, and never want to do another film again!"[20]

http://en.wikipedia.org/wiki/Vivien_Leigh

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Another actress with TB:

Renée Adorée (September 30, 1898 – October 5, 1933) was a French actress who had appeared in Hollywood silent movies during the 1920s.


Tuberculosis and untimely death

By the end of 1930, she had appeared in forty-five films, the last four of them talkies. That year she was diagnosed with tuberculosis, Adorée lived only a few years longer.

Adorée went against her physician's advice in completing her final film, Call of the Flesh, with Ramon Novarro. At the completion of the film, she was rushed to a sanitarium in Prescott, Arizona, where she lay flat on her back for two years in an effort to regain her physical health. In April 1933 she left the sanitarium. At this point it was thought she had recovered sufficiently to resume her screen career. However, she almost immediately grew weaker and her health declined day by day. She was moved from her modest home in the Tujunga Hills to the Sunland health resort in September 1933.[3]

She died there, a few days after her 35th birthday, on October 5, 1933 in Tujunga, California. She is interred in the Hollywood Forever Cemetery, Hollywood, California.

http://en.wikipedia.org/wiki/Ren%C3%A9e_Ador%C3%A9e


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Article mentions that Tay-Sachs disease may help Ashkenazi's prevent TB infections....

http://www.genetics.com.au/pdf/factsheets/fs35.pdf

There are several theories as to why the frequency of the
condition, and the number of genetic carriers for TSD, is high in
the Ashkenazi Jewish population.

• Once a mutation occurs in a gene of an egg or sperm cell, it is
passed down through the generations of a family
• The preference for marrying other Jews would have kept the
faulty HEX A gene within the Jewish population. There would
be a higher chance of an Ashkenazi Jewish couple having a
child with TSD as they would both have an increased chance
of being genetic carriers for TSD

• Another theory is that it is thought that being a genetic carrier
for TSD provides protection against tuberculosis, an infectious
disease that affects the respiratory system. When people
live in overcrowded, difficult circumstances like ghettoes, as
occurred in Jewish history, tuberculosis would have spread
uncontrollably. Those who were genetic carriers for TSD
would have had a better chance of surviving tuberculosis and
have children